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This results in increased AREs expression and proliferation of glutathione (GSH), heme oxygenase-1 (HO-1), superoxide dismutase (SOD), and NAD(P)H: As demonstrated in the relevant literature, quinone oxidoreductase 1 (NQO1) (Xia et al

It is urgent to further explore the mechanisms that regulate ferroptosis in different kidney diseases and determine how to effectively regulate ferroptosis

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