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Under pathological conditions, disruption of TFAM can lead to mtDNA mispackaging, a massive release of damaged mtDNA, and down-regulation of TFAM also reduces the stability of the mtDNA mimetic nuclear structure (West et al., 2015)

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These changes in intestinal-level metabolic processes due to lack of gut microbes are reflected in the altered extra-intestinal metabolism and neurological phenotype 12,13

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