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November 15, 2013

This is consistent with other studies that have implicated the involvement of the mitochondria-dependent intrinsic apoptotic pathway through an NF-B-regulated, mitochondria-mediated pathway involving the activation of reactive oxygen species [307, 308] and the promotion of endoplasmic reticulum stress-mediated mitochondrial apoptosis via the IRE1/ASK1/JNK/Bcl-2 family protein pathways [308]

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