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During the study, patients with intestinal inflammation who were given NAC experienced less inflammation, reduced oxidative stress, an improvement in energy levels, and repair to intestinal tissue damage

Manov I, Hirsh M, Iancu TC (2002) Acetaminophen hepatotoxicity and mechanisms of its protection by N -acetylcysteine: a study of Hep3B cells

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However, this result is expected considering the very high activity of glutaredoxins as deglutathionylating enzymes and a decreased GSSR concentration neither excludes mechanism (ii) or (iii) from Fig