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So distribution and fate are governed more by whole-body copper homeostasis and plasma copper-binding proteins (ceruloplasmin and albumin) than by the peptide acting as an independent drug

Sci Adv 7 (1):eabc7409 Ngo V, Duennwald ML (2022) Nrf2 and oxidative stress: a general overview of mechanisms and implications in human disease

It is hypothesized that eryptosis in patients with HB-ACLF is, at least in part, stimulated by specific components in the plasma, causing accelerated eryptosis and contributing to the development of anemia [250]

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