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Intestinal dysbiosis, by increasing inflammation, can alter the tryptophan pathway with increased indoleamine 2,3-dioxygenase (IDO) activity that reduces the tryptophan metabolism to serotonin and increases the kinurenine pathway with overproduction of quinolinic acid (QA), which has inflammatory effects in the brain [105]

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In addition, astaxanthin increased gene expression of the nuclear factor erythroid 2-related factor 2 (Nef2), Heme oxygenase-1 (HO-1) and NAD(P)H quinone oxidoreductase 1 (NQO1), activating the Nrf2- antioxidant response element (ARE) signaling pathway