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Tamagawa-Mineoka R, Katoh N, Kishimoto S

BPC-157's ability to downregulate this pathway in animal models suggests a mechanism that addresses diabetic motility dysfunction at the neuronal level

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50 & Target Kd: 65 pM (HGF) [1] In Vitro Dihexa binds with high affinity to HGF and both dihexa and its parent compound Norleucine 1-AngIV induce c-Met phosphorylation in the presence of subthreshold concentrations of HGF and augment HGF-dependent cell scattering