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Taufocused therapies, such as aggregation inhibitors and antitau antibodies, aim to prevent neurofibrillary tangle formation (Zempel et al

Repair proceeds through overlapping phases: hemostasis, inflammation, proliferation (angiogenesis, fibroplasia, matrix deposition), and remodeling (maturation and cross-linking of the matrix)

This discovery prompted scientists to investigate whether a similar mechanism might play a role in RA

This process can help maintain redox balance (the steady state between oxidation and antioxidant protection)