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In the last 10 years, discovery of BCL11A , a major -globin gene repressor, has led to a better understanding of the switch from fetal to adult hemoglobin and a resurgence of efforts on exploring pharmacological and genetic/genomic approaches for reactivating fetal hemoglobin as possible therapeutic options

doi: 10.1093/jpp/rgaa036 14 WangSHuaYXuLZouLLiuXLuoYet al

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At the same time, the proteomic profiling gave the opportunity to monitor the responses of infected Calu-3 cells after allicin treatment

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