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More direct evidence for the translocation of zinc was provided by the findings of Koh and others (11), who demonstrated that during a brief period of global ischemia, intracellular zinc accumulation in vulnerable CA1 pyramidal hippocampal neurons preceded degeneration, which could be prevented with the intracerebroventricular administration of the high affinity, membrane-impermeable zinc-chelator, ethylenediaminetetraacetic acid (EDTA) saturated with calcium (Ca-EDTA)

Koblish HK, Zhao S, Franks CF, Donatelli RR, Tominovich RM, LaFrance LV et al

Extra-cellular matrix rigidity may dictate the fate of injury outcome

By topping off your levels early, you provide your cells with the antioxidant support they need to handle the day's challenges

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