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We showed that the PfGrx-dependent oxidation and reduction of roGFP2 both efficiently occur via a monothiol mechanism (Fig

These peptides are claimed to support tissue regeneration, reduce inflammation, speed up recovery of muscles, tendons, ligaments, and soft tissues, and sometimes even serve as alternatives to surgery when healing is stalled

This activation leads to the proteolytic breakdown of DNA repair proteins, cytoskeletal proteins, and caspase-activated DNase inhibitors, ultimately culminating in morphological changes and DNA damage characteristic of apoptosis

10.3390/v12091008 [DOI] [PMC free article] [PubMed] [Google Scholar] Su L., Qu H., Cao Y., Zhu J., Zhang S

2) the transplantation of astrocytes and/or neuroprogenitor cells (NPC) overexpressing Nrf2