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The nuclear translocation of activated AKT induced the phosphorylation of FoxO proteins which evoked the 14-3-3-mediated export of FoxO proteins from the nuclei

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Conversely, exogenous MSC-Exos have emerged as promising therapeutic tools to suppress ferroptosis in diverse diseases, as will be discussed in the later sections

Muscle Healing In BPC 157 studies, in addition to the myotendinous junction (dissection) [43], muscle lesions were induced by transection, contusion, corticosteroid application, and nerve transection [44,45,46,47,48]