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On the other hand, kallistatin and transferrin may be more directly related to changes in condition, as they are involved in the regulation of oxidative stress and are therefore positioned within downstream processes

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This multi-level regulation keeps glutamate within its tight physiologic range. Excess causes excitotoxic neuronal death (as in stroke), while deficiency impairs learning and memory

These experiments suggest that wtIDH1 is an important enzymatic node that influences the flow of carbon into and through the TCA cycle, as summarized in Fig

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