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Treatment with an antagonist of the EP2 receptor (a downstream regulator of PTGS2) caused a significant reduction in the seizure frequency, incidence and duration of seizures, alluding to the role of PTGS2 46

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Moreover, blocking the L-type VSCCs severely weakened the repression of H2AK119ub levels by BHBA, indicating that the BHBA-triggered activation of L-type VSCC-related calcium signaling resulted in the downregulation of H2AK119ub in hippocampal neurons (Figure 5D)