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20 , e10361 (2018)

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This figure is a lot better than 0% of subjects being smoke-free after a year, however an efficacy of 9% leaves a lot of room for improvement

We can now begin to decipher which cell types, cellular functions and genes contribute to smoke-induced injury in normal lung, as well as during COPD exacerbations in individual patients, and thereby, identify common as well as patient-specific disease factors. The Washington Times report indicates that the device described by the Harvard researchers enables them to simulate the actual intaking of toxic smoke without complications related to testing either humans or lab animals

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