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This is likely due to structural differences: ipamorelin Aib/D-2-Nal backbone produces a different binding mode at GHS-R1a that may favor specific downstream signaling pathways (biased agonism) compared to the His/D-Trp backbone of GHRP-6

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In the MenSC-luc, ove-ECM1-MenSC-luc and sh-ECM1-MenSC-luc treatment models, no significant differences were observed among the cell biodistributions across these groups (Additional File 5 Fig

J.MuijtjensM.KobayashiK.TakanoR.KannoS

Side effects, such as blood-borne infections, are still possible but the risk of side effects is mitigated when the platelets are autologous