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Although ferroptosis has been implicated in the pathogenesis of some diseases, such as cancer, ischemic organ damage, neurodegeneration, and pulmonary fibrosis [12], limited mechanistic studies have focused on ferroptosis in EMs-related infertility

Observed outcomes include specific cellular and molecular endpoints such as: Enhanced fibroblast migration capacity Increased collagen and elastin deposition Modulation of pro-inflammatory and antioxidant mediators Altered transcriptional profiles related to tissue homeostasis, repair signaling, and stress response All reported observations derive exclusively from preclinical laboratory and animal studies and remain model-specific

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