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The function of this cycle, proposed by Alton Meister [10], is the transfer of amino acids, finally reaching GSH synthesis [11]

Rapid depletion of endogenous UA reserves during severe ischemia leads to the failure of the natural antioxidant defense system, triggering the need for urgent exogenous UA supplementation for cerebral protection during acute stroke [150]

Scientific References Long R3 insulin-like growth factor-1 (IGF-1) stimulates muscle cell hypertrophy Pharmacokinetics and tissue distribution of IGF-1 LR3 in preclinical models Long R3 insulin-like growth factor I (IGF-I) stimulates protein synthesis in muscle cells IGF-1 LR3: a potent analog with reduced binding to IGF-binding proteins