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Herein, we provides a narrative review based on previous literature that provides a quick overview of generation of ROS, associated diseases and their inhibition by antioxidants

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To explain the similarities and differences in the phenotypic characteristics associated with DS, a gene dose imbalance theory has been hypothesized stating that patients with DS have an increased dose or number of gene copies on Hsa21, which may lead to increased gene expression

During susceptible interactions, not only is glutathione content and distribution changed but there are also changes in the expression of genes encoding specific enzyme changes ( Figure 2 )