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A consistent finding across this body of work is BPC-157s capacity to accelerate healing timelines and reduce inflammatory markers, effects attributed to its interaction with the NO system (both the constitutive NOS and inducible NOS pathways) and its promotion of angiogenesis through upregulation of VEGF expression

BPC-157 accelerates repair, but mechanical stimulus (controlled loading) optimizes the quality of healing

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And this "may slow the accumulation of plaque in the arteries," he says

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