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Central mechanisms driving this secondary damage include vascular disruption and ischemia, excitotoxicity mediated by excessive glutamate, oxidative stress, lipid peroxidation, and the release of proinflammatory cytokines and signaling molecules, which create a pro-inflammatory microenvironment (Eftekharpour et al., 2008

At present, ferroptosis was often regarded as a detrimental factor in certain liver diseases, ferroptosis-inducing agents were mainly used to treat HCC and liver fibrosis (28)

The percentage of activated T-cells (CD4 + CD25 + ) ( Figure 6E ) relative to total CD4 + increased slightly until 0.5 h and then decreased sharply by 81% to a nadir at 9 h pi

PubMed Jastreboff, A.M., Aronne, L.J., Ahmad, N.N., Wharton, S., Connery, L., Alves, B., et al