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Based on these findings, in women with prolactinomas after menopause, besides the common higher risk of developing cardiovascular diseases as compared to fertile age, a further increase in cardiovascular risk might depend on the metabolic impact of PRL excess, thus suggesting a greater risk of a cardiovascular event in these patients as compared to healthy postmenopausal women

Future studies are required to understand why axonal fusion is specifically induced by ferroptosis signaling

Published sensitization studies report rates below 1% in patch testing panels, and no carcinogenic or reproductive toxicity signals have emerged in long-term surveillance data

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