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Redox reactions typically utilize the biologically accessible oxidization states of the metal core to transfer electrons to naturally occurring substrates that are essential for the cancer cell metabolism and therefore induce cell death

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The same reaction can occur on protein cysteine, and the formed GSNO group functions as important intermediate in NO metabolism, a critical player in fulfilling the biolgogical function of NO in signal transduction ( S -glutathionylation (Zeng et al., 2001) or thanks to catalytic activity of GGT generate S-nitrosocysteinylglycine that in turn can be cleaved by a membrane dipeptidase thus forming S -nitrosocysteine (S-NO)