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Recent reports suggest that lung macrophages are important orchestrators of the termination and resolution of inflammation and initiate parenchymal repair processes that are essential for a return to homeostasis with normal gas exchange [14, 15]

GSH is the essential co-substrate for the glutathione peroxidase (GPx) family including GPx4, which converts toxic lipid peroxides into non-toxic alcohols and is central to ferroptosis research and for glutathione S-transferases (GST)

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