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Cellular senescence leads to the upregulation of the p53/p21 and p16/RB signaling pathways and increased expression of enzymes such as beta-galactosidase

MOTS-C is a mitochondrial-derived peptide that influences energy metabolism

(4) After cell stress or injury, these cells are posited to activate innate immune pathways, especially Toll-like receptor (TLR) signaling, which may drive inflammatory responses inside the cell