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These ligands can be proteins, peptides, antibodies, or other biomolecules that have a high affinity for surface markers or receptors that are uniquely expressed by harmful bacteria, as shown in Fig

The selective autophagy substrate p62 activates the stress responsive transcription factor Nrf2 through inactivation of Keap1

Only with a clinician, given the limited evidence and narrowed access after FDA restrictions

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The mushroom toxin, phalloidin binds to actin thus disrupting the cell cytoskeleton, resulting in increased plasma membrane permeability [57]