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TRIB3-deficient cells exhibit elevated CHAC1 levels, leading to increased GSH consumption and higher susceptibility to arsenite toxicity

Interestingly, changes in calcium homeostasis in the brain may contribute to the cognitive decline associated with normal aging and possibly to the development of neurodegenerative disorders (61, 62)

Additionally, GSTs possess the role of facilitating reactions concerning protein S-glutathionylation, where proteins like peroxiredoxin-VI, p53, and protein disulfide isomerase are usually substrates for protein S-glutathionylation mediated by GSTs