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[DOI] [PubMed] [Google Scholar] 87.Kumar A., Dhull D.K., Gupta V., Channana P., Singh A., Bhardwaj M., Ruhal P., Mittal R

Moreover, the virus-induced gene silencing of SlGR3 turned the tolerant cultivar into a susceptible one

Figure 2 Atherosclerosis, a chronic inflammatory disease, may be an ideal environment for the high viral replication capabilities of SARS-CoV-2 in human cells, enhancing hyper-inflammation secondary to immune system dysregulation (Figure 3) that leads to adverse outcomes, as shown in patients with cardiovascular risk factors

1 Like most complex genomic regulatory systems, Nrf2 (nuclear factor erythroid 2-related factor 2) is sequestered in the cytoplasm until it is able to translocate into the nucleus, where it forms a heterodimer with MAF (another transcription factor) and binds to the promoter region of genes containing an antioxidant response element (ARE) sequence

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