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The I/R-AKI mechanism involves a complex series of interactions among inflammatory components, including oxidative stress, degradation of lipids through peroxidation, and reactive oxygen species generation, initiating an inflammatory sequence that results in cellular destruction and renal tissue damage (Shiva et al., 2020

Cell Metab, 2013

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GSTs, as phase II detoxification enzymes, have a widespread distribution across parasite tissues, potentially accounting for up to 3% of soluble cytosolic proteins in cestodes (Brophy and Barrett, Reference Brophy and Barrett1990 a )