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Tau-driven 26S proteasome impairment and cognitive dysfunction can be prevented early in disease by activating cAMP-PKA signaling

The scientific community recognises that peptides, by their nature, are susceptible to degradation by heat, moisture, and light

Critically, this mechanism is selective for senescent cells because non-senescent cells do not accumulate FOXO4 in PML bodies and do not depend on FOXO4-p53 sequestration for survival

These effects are usually temporary and subside within a few hours

Together, these findings indicate that melatonin drives ILC2s into a metabolic state incompatible with a pro-inflammatory effector phenotype