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Gene alterations modulating glutamine dependency in cancer The dependency of glutamine metabolism in cancer is not simply a passive adaptation to proliferative state, but rather a tightly regulated response to a multitude of intracellular and extracellular factors, including potential genetic alterations

Statistical significance was calculated by two-way ANOVA followed by multiple comparisons in a-c, f , and g and unpaired two-sided t-test in e

L-carnitine is, therefore, bestowed to oversee the proper functioning of the heart and blood vessel systems

The venom optimization hypothesis revisited

Mitochondrial DNA in inflammation and immunity